Serum uric acid (SUA); cardiovascular disease (CVD); coronary heart disease (CHD); endothelial nitric oxide (eNO); endothelial nitric oxide synthase (eNOS); endothelial nitric oxide (eNO); reactive oxygen species (ROS); metabolic syndrome (MS); insulin resistance syndrome (IRS); nicotine adenine dinucleotide phosphate oxidase reduced NAD(P)H; superoxide (O2-•); xanthine oxidase (XO); type 2 diabetes mellitus (T2DM); angiotensin converting enzyme (ACE); renin-angiotensin-aldosterone system (RAAS); advanced glycosylation endproducts (AGE); superoxide dismutase (SOD); glutathione (GPX); plasminogen activator inhibitor (PAI-1); angiotensin II (AngII); low density lipoprotein cholesterol (LDL-C); asymmetrical dimethyl arginine (ADMA); highly sensitive C reactive protein (hsCRP); national cholesterol educational program adult treatment panel III (NCEP ATPIII); nuclear transcription factor (NF-kappaB); monocyte chemoattractant protein-1 (MCP-1); tumor necrosis factor alpha (TNF alpha); interleukin one beta (IL-1beta); interleukin 6 (IL-6); body mass index (BMI); high density lipoprotein (HDL); hydrogen peroxide (H2O2); free fatty acids (FFA).



A part of this study was supported by NIH grants HL-71010 and HL-74185.

The authors would like to acknowledge Dr. Charles Kilo and Dr. Joe Williamson of Washington University School of Medicine for their devotion to teaching medical students, residents, fellows and patients with diabetes in the pursuit of knowledge regarding diabetes. Their research and 31 years of providing CME exposure through a nationally recognized annual CME Diabetes Symposium have been an inspiration to all interested in delivering the best diabetic care possible to their patients. 

1Department of Family and Community Medicine, University of Missouri, Columbia, Missouri USA
2Department of Physiology and Biophysics, University of Louisville, School of Medicine, Louisville, Kentucky USA

Nutrition & Metabolism 2004, 1:10     doi:10.1186/1743-7075-1-10

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